Brogidirsen, an investigational exon 44 skipping agent for the treatment of Duchenne muscular dystrophy: Clinical trial design (Phase 2)


Topic:

Clinical Trials

Poster Number: M175

Author(s):

Paula R. Clemens, MD, University of Pittsburgh, Pittsburgh, USA, Michelle L. Previtera, PhD, NS Pharma, Inc., Paramus, NJ, USA, Robert A. Crozier, PhD, NS Pharma, Inc., Paramus, NJ, USA, Leslie Magnus, MD, NS Pharma, Inc., Paramus, NJ, USA, Eric Hoffman, PhD, AGADA Biosciences, Inc., Hirofumi Komaki, MD, PhD, Translational Medical Center, National Center of Neurology and Psychiatry, Tokyo, Japan, Yoshitsugu Aoki, MD, PhD, National Center of Neurology and Psychiatry (NCNP), Vamshi K. Rao, MD, Division of Neurology, Ann and Robert H. Lurie Children’s Hospital of Chicago, Chicago, IL, USA

Background: Brogidirsen (NS-089/NCNP-02) is being studied for the treatment of Duchenne muscular dystrophy (DMD) in patients with a dystrophin gene mutation amenable to exon 44 skipping. Brogidirsen is a novel phosphorodiamidate morpholino oligomer (PMO) that contains two linked sequences targeting two separate binding sites within exon 44. In a previous Phase 1/2 study, skeletal muscle dystrophin levels increased significantly after 24 weeks of brogidirsen treatment (mean change from baseline: 13.1%, p = 0.03). This first-in-human data led to FDA orphan drug designation.

Objectives: We present the study design for a Phase 2, open-label, 2-part study (NCT05996003) assessing the efficacy, safety, tolerability, and pharmacokinetics of brogidirsen in ambulant boys with DMD. The primary objectives of Part 1 and Part 2 are to evaluate the safety, tolerability, and pharmacokinetics of brogidirsen. The primary objective of Part 2 also includes dystrophin protein induction in skeletal muscle of participants treated at the maximum tolerable dose (MTD) from Part 1.

Methods: Brogidirsen will be administered weekly by intravenous infusion to ambulant boys aged ≥4 to <15 years with DMD mutations amenable to exon 44 skipping. The study (N = 20) will consist of 2 cohorts. Cohort 1 (n = 6) will receive ascending doses consisting of a 4-week Treatment Phase at each of three dose levels (Part 1). These participants will continue into a 24-week Treatment Phase at the MTD from Part 1 (Part 2). Cohort 2 (n = 14) will go through a 24-week Treatment at the MTD of Part 1 (Part 2) and will undergo muscle biopsies at baseline and Week 25 to determine dystrophin induction. Secondary outcomes of muscle strength and motor function will be evaluated. Conclusions: The design of this 2-part Phase 2 study was informed by the results of the previous Phase 1/2 trial that showed brogidirsen induced significant dystrophin production. The Phase 2 study has begun recruitment.